The story of a lot of women’s hair sees the event of getting their hair too short in the process of trimming split ends and then the hair just stop growing beyond that length. The length of the hair being a major factor of body image in women, such events may be greatly distressing. It is not always because of aging, it is because there is something wrong with your normal hair growth cycle that you may be having telogen effluvium.
What Is Telogen Effluvium?
Hair loss is an understudied subject. However, so far medical researchers have identified about a dozen different types of hair loss. Telogen effluvium is one type of hair loss, has to do with hair growth cycle.
There are three distinct stages of hair growth cycle.
- The first stage is anagen, in which a hair produces from a hair follicle, and grows in length, the stage continues for 3-6yeears.
- After this period, there comes the catagen stage, wherein hair follicles are cut short of blood circulation, while it keeps pushing upwards, without the blood circulation the hair grows dull and sheds off in 2-3weeks.
- Then the telogen stage comes, wherein the hair follicles rest for 2-3monts, before a new hair grows again with the anagen stage.
Telogen effluvium occurs when the resting phase prolongs more than it should. This is usually an autoimmune disorder, meaning it resolves on its own. However, permanent hair shedding including patches of baldness in the vertex region of the scalp might appear in severe cases.
What Causes Telogen Effluvium?
The causes of telogen effluvium is deemed to be a metabolic distress, and considered to be a non-androgenic hair loss type. Excess stress, sebum accumulation, malnutrition and lack of sleep may be the causes of it. Pregnancy and child birth is deemed as one of the major reasons why telogen effluvium is common in women. Other women specific causes include menopause.
Hair loss in women differs from men in many ways. Where men see a patterned baldness with a specific donor reserve, women face a diffuse hair thinning all over the scalp that does not lead to baldness.
However, this characteristic is distinct to androgenic alopecia in which the patient is usually a male, with the front and center of their scalp seeing varying loss of density and baldness, while the back of the scalp remains intact. This happens due to a reaction of androgens that convert into a steroid called dehidrotestosterone (DHT) and inflame hair follicles in the front and central of the scalp.